Abstract

Previous studies have shown that diabetics make tritan-like errors in color-matching experiments^1,2 and demonstrate elevated increment thresholds for test lights detected by an S-cone mechanism^3,4. While these experiments indicate the presence of a defect in visual pathways that receive an input from S-cones, they are unable to identify which components within these pathways are responsible for abnormal visual functions associated with diabetes. The purpose of this study is determine if diabetes introduces an anomaly either prior to and/or at the sites of cone interaction, or after the pooling of cone signals within chromatically-opponent pathways.

© 1990 Optical Society of America